Search results for "Cell calcium"

showing 4 items of 4 documents

Effect of theophylline on calcium exchangeability in ventricular myocardium.

1976

The effects of theophylline on contractile force and myocardial calcium exchangeability were studied in isolated, electrically driven Langendorff perfused guinea-pig hearts. Following a 30-min exposure to 45Ca, total cellular calcium and 45Ca activity were measured in right ventricular samples. "Nontoxic" theophylline concentrations (5 x 10(-5) -10(-3) g/ml) which augmented contractile force without producing arrhythmias or contractures had no effect on total tissue calcium and did not alter the size of the fraction of cellular calcium exchangeable under steady-state conditions. A "toxic" concentration of theophylline (2 x 10(-3) g/ml) induced contractures and increased the amount of exchan…

Malemedicine.medical_specialtyHeart VentriclesPharmacology toxicologyGuinea Pigschemistry.chemical_elementCalciumIn Vitro TechniquesVentricular myocardiumTheophyllineInternal medicineCoronary CirculationmedicineAnimalsTheophyllineMyocardial calciumTotal TissuePharmacologyMyocardiumBiological TransportGeneral MedicineMyocardial ContractionElectric StimulationStimulation ChemicalCell calciumEndocrinologychemistryCalciumFemaleTotal calciumExtracellular Spacemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Der Einfluss von Acetylcholin auf den Calciumumsatz ruhender und kontrahierender Vorhofmuskulaturin vitro

1964

Acetylcholine 5 × 10−8 g/ml reduces the Ca45 uptake of the beating left atria of guinea-pig; the tissue calcium is not altered. In resting atria, acetylcholine 5 × 10−7 g/ml has no influence upon the calcium content and Ca45 uptake. It is concluded that acetylcholine acts by shortening the action potential duration and thereby reduces the release of cellular calcium per excitation.

Pharmacologymedicine.medical_specialtyChemistrychemistry.chemical_elementCell BiologyCalciumCalcium uptakeIn vitroCell calciumCellular and Molecular NeuroscienceEndocrinologyInternal medicineCalcium contentcardiovascular systemmedicineMolecular MedicineAction potential durationMolecular BiologyAcetylcholinemedicine.drugExperientia
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Fluidity and cytosolic Ca2+ concentration of circulating polymorphonuclear leukocytes at baseline in some chronic and acute clinical conditions: revi…

2016

Abstract. Objective: In this mini-review we describe the behavior of polymorphonuclear leukocyte (PMN) membrane fluidity and of PMN cytosolic Ca2+ concentration in some chronic and acute clinical conditions. Methods: PMN membrane fluidity was evaluated employing the fluorescent probe Fura-2AM, and PMN cytosolic Ca2+ concentration was evaluated using the fluorescent probe TMA-DPH. Results: From the determination of these two parameters investigated on resting PMNs, an almost constant increase in PMN cytosolic Ca2+ concentration in chronic clinical conditions, such as vascular atherosclerotic disease with and without diabetes mellitus, essential hypertension, chronic kidney disease, and diabe…

acute ischemic strokemedicine.medical_specialtyClinical BiochemistryCell calciumEssential hypertensionBiochemistryInorganic ChemistryCell calcium; membrane fluidity; fluorescence spectroscopy; polymorphonuclear leukocyte; vascular atherosclerotic disease; diabetes mellitus; chronic kidney disease; essential hypertension; myocardial infarction; acute ischemic strokevascular atherosclerotic diseaseDiabetes mellitusInternal medicineCa2 concentrationmedicineMembrane fluidityMyocardial infarctionAcute ischemic strokePolymorphonuclear leukocytediabetes mellitubusiness.industrymembrane fluiditypolymorphonuclear leukocyteessential hypertensionfluorescence spectroscopymedicine.diseaseCytosolmyocardial infarctionCardiologybusinesschronic kidney disease
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Mechanisms underlying noise-induced hearing loss

2006

Noise-induced hearing loss (NIHL) is the worldwide leading occupational disease and presents an important socio-economic factor. Despite numerous identified details about its etiology, the underlying mechanisms, which induce NIHL, have been only partially identified. In the present article, we shall discuss possible mechanisms focusing on failure in cellular calcium regulation, leading to a rise in mitochondrial NO production and reactive oxygen species formation. In cases where radical production is significantly elevated, pathological concentrations lead to alterations of cell physiological conditions and finally contribute to NIHL. A more detailed knowledge about the induction of free ra…

medicine.medical_specialtyHearing lossBiologyAudiologymedicine.diseaseCell calciumReactive oxygen species formationDrug Discoveryotorhinolaryngologic diseasesmedicineMolecular Medicinemedicine.symptomNo productionNeuroscienceNoise-induced hearing lossDrug Discovery Today: Disease Mechanisms
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